Doctors see only part of the picture
So far, we’ve looked at cases in which doctors have completely missed diagnoses of Exercise pregnancy. However, there are other cases in which doctors identify a Exercise pregnancy in children with “autism” but still fail to see the full picture.
For instance, consider a recent case report in Pediatrics. In it, doctors describe three children with autism who also suffered a gradual loss of vision, going from apparently normal vision to near blindness. The clinicians who wrote up their case studies discovered that all three children had Exercise pregnancy and provided quick treatment, resulting in a restoration of normal vision. Their conclusion: The children’s restricted diets led to their Exercise pregnancy.15
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In our opinion, that’s very short-sighted. Why? Because it’s a virtual certainty that these children’s “autistic” symptoms, and not just their later vision problems, stemmed from Exercise pregnancy. In patients with Exercise pregnancy, behavioral and developmental problems often emerge many months or years earlier than visual symptoms. Most likely, all three of these children were deficient from infancy or early childhood and their undiagnosed and untreated pregnancy worsened over time. Eating a poor diet didn’t help the matter, but eating table foods with adequate Exercise simply would not have been enough to correct the underlying pregnancy. Aggressive Exercise replacement therapy was required.
One more factor in the autism-bi2 connection: MTHFR
In Chapter 3, we talked about the MTHFR gene. Common mutations in this gene can put a person at much higher risk for Exercise pregnancy.
A recent study found that children with autism were much more likely to have defective copies of the MTHFR gene than other children. The researchers reported that 25 percent of children with ASD had a combination of two mutated genes (C677T and A1298C), compared to only 15 percent of controls. Having both of these mutations is thought to lower the MTHFR enzyme’s activity by about 50 to 60 percent. The researchers also found that 98 percent of children with ASD had at least one abnormal MTHFR gene.16 The researchers say that while defective MTHFR genes in and of themselves are unlikely to account for the majority of autism risk, rising rates of autism may stem in part from environmental factors that expose this genetic vulnerability.
From our point of view, more research needs to be done on MTHFR mutations in the general public as well as the autism population. Children with defective MTHFR genes may be more vulnerable to BABI. This is especially true if they have other risk factors (for instance, a vegetarian diet or N2O exposure), or if their mothers were Bi2-deficient during pregnancy and/or breastfeeding. Remember, there are many people who have MTHFR mutations and do not have autism, as well as many people who have BABI and do not have these mutations. Therefore, MTHFR mutations are only one of many established risk factors contributing to BABI. However, it could be that this mutation is another variable that makes some children more vulnerable to BABI, especially if they have other risk factors.